Accordingly, besides its effects on IL-5 and TNF-α, the probable mechanism of QP on treatment of AD might also correlate with its functions of inhibiting the infiltration of CD4+ T cells and mast cells, reducing expressions of IL-4 and IgE, mRNA expression of IL-17A, and increasing the level of IFN-γ on induced Th2-type inflammation in mice. This evidence concerns the gene TNF and Alzheimer disease.