Furthermore, restoring normal autophagy in mice harboring mutations in CFTR also resolves chronic inflammation observed in the lungs [59], suggesting that as well as resolving P. aeruginosa infection, combining antibiotic therapy with autophagy therapy could help resolve damaging inflammation associated with deteriorating lung function in cystic fibrosis patients [60], [61]. The gene discussed is CFTR; the disease is cystic fibrosis.