MYT1 and neoplasm: In order to elucidate the radioimmunotherapy molecular mechanisms of the treatment of gastric cancer cells expressing d9-E-cadherin with 213Bi-d9MAb, Seidl et al. [12] quantified 380 gene expression of 213Bi-treated tumor cells and found that 213Bi-induced cell death was initiated by G2 arrest and up-regulation of several genes, including MYT1.