The involvement of IL-17 in the pathogenesis of EAM and dilated cardiomyopathy [58] and its potential involvement in DCM would be analogous to the insult of acute burn that activates leukocyte TLRs, and the resulting production of numerous cytokines/chemokines, including effectors in burn cardiomyopathy [105]–[108]. The gene discussed is IL17A; the disease is dilated cardiomyopathy.