The involvement of IL-17 in the pathogenesis of EAM and dilated cardiomyopathy [58] and its potential involvement in DCM would be analogous to the insult of acute burn that activates leukocyte TLRs, and the resulting production of numerous cytokines/chemokines, including effectors in burn cardiomyopathy [105]–[108]. This evidence concerns the gene IL17A and familial dilated cardiomyopathy.