SLC32A1 and epilepsy: As far as the experimental models of epilepsy are concerned, the density of VGAT terminals is reduced in an animal model of cortical dysplasia (Zhou and Roper, 2010), in pre-seizure seizure-sensitive gerbil (Kang et al., 2003) and picrotoxin-induced kindling rat hippocampi (Jiang et al., 2004), although VGAT-IR is unaltered in KA-induced seizures (Sperk et al., 2003).