In our series, the distribution of tumor molecular subtypes was: 1 (4.2%) estrogen receptor-positive luminal A (case 5 in Table 2, Figure 2A-D), 1 (4.2%) estrogen receptor-positive luminal B (case 4 in Table 2, Figure 3A-D), 1 (4.2%) HER2 overexpression (case 3 in Table 2, Figure 4A-D), and 19 (79.2%) basal-like (Table 2, Figure 1A-D). This evidence concerns the gene ERBB2 and neoplasm.