IL2 alone conferred detectable resistance to TB lesions compared to BSA/saline, but could not effectively reduce Mtb bacterial burdens([22], and Fig. 1c, Fig. 3a), whereas Picostim/IL2 expansion of Vγ2Vδ2 T effector cells decreased levels of Mtb infection(Fig. 1c). This evidence concerns the gene IL2 and tuberculosis.