Other mutated genes (e.g., WT1, IDH1/IDH2, TET2, RUNX1, and MLL) or aberrantly expressed ones (e.g., BAALC, ERG, EVI1, and miR-181a) will likely become useful in refining molecular risk in CN-AML [4-16]. This evidence concerns the gene KMT2A and acute myeloid leukemia.