CXCL10 and infection: In contrast, significant increases in the levels of the chemokines CXCL9 (MIG) and CXCL10 (IP-10), previously demonstrated to play key roles in the trafficking of lymphocytes to sites of inflammation [24], [25] and known to be associated with clinical allograft rejection [26], [27], were observed in infected animals relative to non-infected mice at 2, 4, and 6 weeks after infection (Figure 2).