CNBP and myotonic dystrophy: In addition to inducing a “sliceopathy,” the repeat expansions may also induce disease through altered function of DMPK and ZNF9, dysregulated expression of neighboring genes, and repeat associated non-ATG translation.33–36 Recent studies have also begun to elucidate the role of repeat expansions in the central nervous system (CNS) manifestations of myotonic dystrophy, including cognitive dysfunction and sleep disorders.37,38