Taking into consideration that in CEL and CML cell lines the situation with E- and especially P-selectin ligands appears to be more complicated than expected from recent literature on granulocytes – which more or less takes the essential role of sialyl Lewis x for granted [42], [43] – we think that our results certainly warrant further examination of the selectin ligands on other cell lines of CEL, CML and other leukemia entities as well. This evidence concerns the gene FUT4 and Chronic Eosinophilic Leukemia, Not Otherwise Specified.