Taking into consideration that in CEL and CML cell lines the situation with E- and especially P-selectin ligands appears to be more complicated than expected from recent literature on granulocytes – which more or less takes the essential role of sialyl Lewis x for granted [42], [43] – we think that our results certainly warrant further examination of the selectin ligands on other cell lines of CEL, CML and other leukemia entities as well. The gene discussed is SELP; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.