Recent reports demonstrated that FOXA1 acted as an AR cofactor to drive G2/M cell-cycle progression [82] and that AR and FOXA1 functionally cooperate and enhance proliferation and migration of prostate cancer cells [80] although there are conflicting reports on FOXA1 function in metastatic progression of prostate cancer [80,83]. This evidence concerns the gene FOXA1 and prostate carcinoma.