In this regard, we previously reported that administration of CAM in influenza A virus (IAV)-infected mice suppressed tumor necrosis factor alpha production and augmented interleukin-12 production in the blood [11], [12], resulting in alleviation of the flu symptoms, while oral treatment with OSV attenuated the induction of respiratory anti-IAV specific secretory IgA (S-IgA) immune responses [3]. This evidence concerns the gene CD79A and influenza.