In recent years, experimental studies in models of cerebral ischemia/reperfusion injury, ischemic stroke, intracerebral hemorrhage, traumatic brain injury and spinal cord injury have revealed a crucial role of PPARγ and its ligands in attenuating neuronal cell death in the injured CNS [19], [20], [21], [31], [32]. The gene discussed is PPARG; the disease is intracerebral hemorrhage.