Both IL-17A and IL-22 have been shown to be pro-inflammatory cytokines that participate in the pathogenesis of autoimmune diseases, such as rheumatoid arthritis (RA) [11], Crohn’s disease [12], systemic lupus erythematosus (SLE) [13], and psoriasis [14]. This evidence concerns the gene IL17A and systemic lupus erythematosus.