SCRIB and neoplasm: Expression arrays and ChIP-Seq analysis of Ab binding regions and immunohistochemical analysis of the tumours indicates that Ab promotes the retention of a progenitor-like cell state in scrib mutant cells by blocking the expression of dac, eya, dan, ato and elav in the eye disc, and prevents the temporal elaboration of cell fate domains, defined by dac, cut (ct), senseless (sens), dan, bab2 and ato expression, along the proximo-distal axis in the antennal disc.