OASL and infection: Among these cells, we showed in our study that the critical cellular source for the sustained and higher serum IFN-I level in Oasl1 KO mice at the early stage of infection (2 d p.i. when the IFN-I level in serum became clearly stronger in the KO mice than in WT mice) was pDCs (CD11cintB220+CD8+/−), although cDCs and Macs of Oasl1 KO mice also produced significantly more IFN-I than those of WT mice (Fig. 5D).