Given that the ineffectiveness of exogenous IFN-I at the later phase of chronic virus infection, it is worthwhile to note that Oasl1 expression in pDCs was still sustained at a later phase (30 d p.i.)of chronic LCMV CL-13 infection but not of acute LCMV Arm infection, whereas Ifna/b1 expression in the pDCs returned to the basal level by that phase in both LCMV infections (Fig. S11). This evidence concerns the gene OASL and viral infectious disease.