IFNG and autoimmune myocarditis: Since NK cells are important effector cells with cardioprotective properties, limiting virus replication [35] through the release of IFN-γ [108], an MSC-mediated repressive effect on NK cells at an early stage of infection when NK cells play an indispensable role in the inhibition of the viral replication [35] could favor viral propagation and exacerbation of autoimmune myocarditis.