While more resistant to the infection in the early stage– likely due to the high levels of IL-22 (Figure S2A in Text S1)–both IFN-γ- and IL-17RA-deficient mice progressively become susceptible to infection, as indicated by the failure to restrain fungal growth during the primary infection (Figure S2B in Text S1) or after re-challenge (Figure S2C in Text S1) and to limit inflammation (Figure S2D-F in Text S1). Here, IL17RA is linked to infection.