Despite both types of mice show a higher fungal burden early in infection, as compared to wild-type mice (Figure 2A), IL-17F-deficient, more than IL-17A-deficient, mice showed signs of inflammation (Figure 2B) and PMN recruitment (Figure 2C and inset in Figure 2B) associated with high levels of IL-17A (Figure 2D). This evidence concerns the gene IL17A and infection.