Given that siderophores exhibit a higher affinity for iron than lactoferrin [12], it is likely that fungal siderophores can acquire iron from lactoferrin during infection; however, the rate of siderophore iron acquisition in a lactoferrin-rich environment is likely slower than in the absence of lactoferrin given the scarcity of free iron or iron-bound to lower affinity biomolecules. The gene discussed is LTF; the disease is infection.