Namely, whilst impairments in the dynamic behavior of retinal microglia in the resting state, and impaired microglial migration following laser injury in CX3CR1 deficient mice have been reported [11], CX3CR1 deficiency does not affect microglial responses to acute light damage [12] or proliferative neovascularization in a mouse model of retinopathy of prematurity [13]. The gene discussed is CX3CR1; the disease is retinopathy of prematurity.