Following Vac-GP infection, WT and bim−/− SMARTA efficiently populated the memory pool with similar efficiency, indicating that the unique role of Bim in regulating cell death of low avidity Lm-gp61-induced SMARTA cells during the contraction phase may rely in part on the nature of the infectious challenge (Fig. 2C and 2D). This evidence concerns the gene BCL2L11 and infection.