An example of this is provided by pro-resolution lipid mediators, such as lipoxins, protectins and resolvins, which were reported as being able to attenuate disease severity in mouse asthma models (Haworth et al., 2011; Haworth et al., 2008; Levy et al., 2005; Levy et al., 2002; Levy et al., 2007a; Levy et al., 2007b). The gene discussed is CD59; the disease is asthma.