CCL2 and metabolic dysfunction-associated steatohepatitis: These findings are in line with previous observations on the role of immunocompetent cells promoting liver injury from apoptosis ligands and in the context of NASH through alternations in intrahepatic T-cell populations.16, 17 Further, we underlined the contribution of proinflammatory cytokines such as CCL2 (MCP-1), IFN-γ and TNF to STZ-induced liver injury.