CAMK2N1 and prostate neoplasm: Other sources of evidence including lncRNAs known to be involved in prostate cancer adjacent to genes comprising GC (e.g. PCAT-113 [33], found 200 bps upstream of CAMK2N1) as well as overlapping copy number alterations found in prostate tumors (e.g. the copy number amplification reported by Taylor et al [73] in chr5p15.2 and PCAT-32) add to the sources of evidence on their association with prostate cancer and involvement in multiple biological processes that must occur for tumorous tissue to leave the prostate bed during the metastatic process.