Mice that received Th2 cells secreted IL-4, IL-5 and IL-9 (Figure 4D) and up-regulated Arg1, Eotaxin (Ccl11) and Gob5 (Clca3) within the lung (Figure 4E), characteristic of Th1 or Th2-mediated airway inflammation. This evidence concerns the gene CCL11 and inflammatory response.