FCGR3A and infection: The expression of the inhibitory receptor FcγRIIB was increased and peaked at 28 DPI, whereas that of the activating receptor FcγRIIIA was decreased and reached the lowest point at 5 DPI in both PRRSV HN07-1 and BJ-4 infections, contributing to the decreased phagocytosis of PMNs and possibly to the suppression of immune responses.