Recent evidences indicate that asthma encompasses many different phenotypes ranging from classical TH2 allergic phenotype, characterized by airway eosinophilia, to a TH1 or TH17 dominated phenotypes, which are correlated with airway neutrophilic inflammation and production of IFN-γ and IL-17, respectively [2], [3]. This evidence concerns the gene IL17A and Increased total eosinophil count.