In our previous study, inflammatory agents including bacterial endotoxin lipopolysaccharide (LPS) and proinflammatory cytokines interleukin-1 (IL-1) and TNF, induced CXCL1-3 and CXCL8 in ovarian cancer cells by involving NF-κB signaling[10]. This evidence concerns the gene NFKB1 and ovarian cancer.