Other members of the family such as APRIL (TNFRSF13B) and BAFF (TNFRSF17) were shown to be involved in the protection of MM cells from apoptosis via NF-κB activation [9], while loss-of-function mutations of Fas antigen (TNFRSF6) could inhibit Fas ligand induced apoptosis in MM cells [10]. This evidence concerns the gene FASLG and Miyoshi myopathy.