Alterations of iron status have been described in child obesity [1,2,3,4,5,6,7,8,9,10]; inflammation is associated with a shift of Fe from blood, decreasing serum Fe and transferrin levels; so when they are used as surrogate markers of Fe status, an increased prevalence of deficiency is found in overweight subjects [1,6,9]; on the other hand ferritin is usually normal or increased in these patients [3,9,10]; measurement of other parameters of Fe metabolism such as hepcidin did not contribute to the diagnosis [7,10]. This evidence concerns the gene TF and hyperinsulinemic hypoglycemia, familial, 4.