One study attributed the feeble LCMV-specific NAb responses to an instrinsic property of the LCMV glycoprotein itself as infection of mice with recombinant LCMV expressing the VSV-glycoprotein generated robust and rapid NAb responses similar to the wild-type (WT)-VSV while infection of mice with recombinant VSV expressing the LCMV glycoprotein displayed delayed kinetics and very low titers of NAb responses similar to WT-LCMV [30]. The gene discussed is ART4; the disease is infection.