Although inflammation appears to play a crucial role in both PCA [1] and BPH [2], our results support the observation that the inflammation associated with BPH consists largely of IL-15 and γ-interferon recruited CD4+ T-Lymphocytes [2], and it contrasts with the tumor-associated macrophages present in the tumor microenvironment. The gene discussed is CD4; the disease is benign prostatic hyperplasia.