IFNG and infection: In contrast to the induction of an atypical population of infection-induced myelo-lymphoid progenitors, which we showed to be critically dependent on the presence of IFN-γ receptor on HSCs/HPCs for their emergence [8], the effect of IFN-γ on the contraction of BM myelopoiesis was mediated by an irradiation-insensitive cellular compartment.