In addition, the myeloid competence of multipotent progenitors (LMPP) and myeloid restricted subsets (CD27+ CMP and GMP) was not altered in the absence of IFN-γ receptor before and during acute malaria, excluding a change of developmental potential in response to lack of IFN-γ signaling in steady-state or after infection (Figure S5). This evidence concerns the gene IFNG and infection.