CD1D and Miyoshi myopathy: It has also been previously described [26] that loss of CD1d, the lipid presenting molecule, could be the reason for the loss in iNKT activity, therefore we followed its expression during the course of the disease, both on total spleen and BM cells, containing the APCs and also on the 5T33MM cells as it has been described that MM cells can function as APCs themselves [28].