GATA2 and infection: It is highly likely that histones are also recruited to latent promoters immediately post infection and, akin to the binding of ERF and YY1 to the MIEP resulting in the recruitment of histone deacetylases and histone methyltransferases, it is highly plausible that the binding of GATA-2 to the UL144 and LUNA promoters in latently infected monocytes [47] results in the recruitment of, for example histone acetyltransferases, generating the histone signature we observe in latently infected cells [46].