Thus, it can be speculated that antimicrobial peptides LL-37, hBD-3, and HNP-1, in concert modulate neutrophil apoptosis in vivo in the local milieu at the sites of inflammation or infection by utilizing different receptors (e.g., FPRL1, P2X7, CCR6, and P2Y6). The gene discussed is CAMP; the disease is infection.