In a complementary approach, we transiently expressed KCC3, KCC3mPro and KCC3ΔCterm in HeLa cells and, similarly to what was noticed previously, we detected endogenous expression of KCC3 in this cervical cancer cell line [3], as well as differences in protein levels of the wild-type and mutant forms likely due to the C-terminus involvement in the protein stability [3] (Figure 4B and 4C). This evidence concerns the gene SLC12A6 and cervical cancer.