Mesenchymal-like tumor cells gain migratory capacity through abnormal survival signals via receptors such as fibroblast growth factor receptor (FGFR), hepatocyte growth factor receptor (MET), transforming growth factor beta receptors (TGFβRs), insulin like growth factor 1 receptor (IGF1R), platelet derived growth factor receptor (PDGFR), and regulatory kinases such as phosphoinositide-3-kinase (PI3K), v-akt murine thymoma viral oncogene (AKT), and mechanistic target of rapamycin (mTOR) [5]–[10]. This evidence concerns the gene IGF1R and neoplasm.