SEMA3A and myocardial infarction: Cardiac nerve injury caused by MI could also trigger the reexpression of Sema3a and other neurotrophic factor genes in the non-neural cells around the site of injury 34–36, leading to nerve regeneration through nerve sprouting, 9, 37, 38 whereas disturbance on Sema3a expression system was associated with compensatory sprouting and synaptic remodelling 39.