Release of cytokines (IL1β, IL-6, and TNFα), nitric oxide, endotoxins [3], and activation of caspases (caspases 3) [20] in setting of a gram negative bacteremia and sepsis leading to myocardial depression and ventricular dilatation [3] is another theory to explain SRTE (Figure 1). This evidence concerns the gene IL1B and Sepsis.