Consistent with PI3K activation in the viral infection and with Akt being a target of activated PI3K, the extent of Akt phosphorylation was greater in the 0.25 h and 0.5 h post-infection samples than in the corresponding mock-infected control (Figure 2A, “pAkt” and Figure 2C, “Akt”). The gene discussed is AKT1; the disease is viral infectious disease.