The molecular pathways dysregulated by these differentially expressed miRNAs in DHT-treated ovaries was evaluated and included signalling by androgen, aldosterone, VEGF, FGF, growth hormone, 14-3-3, calcium, estrogen receptor, G-Protein coupled receptor, glucocorticoid receptor, and apoptosis as well as in ovarian cancer signalling and androgen and oestrogen metabolism (Table 1). This evidence concerns the gene GH1 and ovarian cancer.