Although Gata3 is known to play a crucial role in T cell development [22,37], our elimination of Il2ra as the basis of the mutant phenotype as well as McElwee et al.’s failure to detect any signs of hair follicle inflammation in jal/jal mutants [10] suggest that mouse juvenile alopecia does not provide an ideal model for AA. Here, IL2RA is linked to alopecia.