As shown in Figure 3, when we analyzed 6 each of RTK, CCSK, ESFT, and NK as well as 21 cases of Wilms’ tumor and 9 cases of CMN, the CpG sites of THBS1 were unmethylated in all of the cases, indicating that the CpG sites of THBS1 are specifically hypermethylated in CCSK among pediatric renal tumors. This evidence concerns the gene THBS1 and Wilms tumor.