Since pituitary PRL secretion may be regulated by IFNγ and TNFα, albeit in rodent studies [46], [47], [48], [49], and since these cytokines [50], [51] and PRL [3], [5], [28], [52], [53] have been implicated in the pathogenesis of psoriasis, we also examined whether these prototypic pro-inflammatory cytokines influence PRL and PRLR expression in the HF. This evidence concerns the gene TNF and psoriasis.