It has also been shown that these molecules participating in the infiltration of inflammatory cells into the airways in allergic inflammation, probably suppressing chemokine and cytokines, such as eotaxin, IL-5 and IL-13, related to the pathophysiology of asthma, subject that will be discussed further below (Taki et al., 2007; Possa et al., 2012). This evidence concerns the gene IL13 and asthma.