Moreover, our previous works have identified an increased expression of both KGFR and FGFR2-IIIc also in pathological events, such as the progression and stadiation of classic Kaposi Sarcoma and dermatofibroma, two tumors involving the dermal compartment characterized by a prevalent inflammatory component [27]–[28] and elevated levels of proinflammatory cytokines [22], [29]. This evidence concerns the gene FGFR2 and Kaposi's sarcoma.