Association of defective glucose disposal with high CD4 counts might be due to post-HAART recovery of CD4 T cells that are unable to utilize glucose effectively because of low GLUT-1 or IL7-R expression or to the influence of genetic factors such as CCL3L1 or CCR5 polymorphisms known to be associated with the development of type 1 diabetes[23]. Here, CCL3L3 is linked to type 1 diabetes mellitus.