BCL2 and pancreatic neoplasm: Li et al demonstrated that maslinic acid, a triterpene derivative obtained from olive pomace, inhibited endogenous NF-κB activity in a time-dependent manner and the TNFα-induced NF-κB activation in a dose-dependent manner in pancreatic cancer cells, and thus prevented the expression of downstream target genes, such as Bcl-2, Survivin, COX-2 and IAP-1 (21).